ABSTRACT. Desquamative gingivitis is a descriptive term of nonspecific clinical expression in the gingiva (redness, burning, erosion, pain) of several. Desquamative gingivitis (DG) is a clinical term used to describe gingival tissues that demonstrate potentially painful gingival erythema, hemorrhage, sloughing. Lichen planus is an idiopathic t-cell mediated inflammatory condition. Although its etiology is unknown OLP is sometimes associated with other medical.

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Blood tinged intact bulla with desquamative gingivitis seen in respect to left maxillary posterior teeth. Diagnoses Chronic periodontitis Localized aggressive periodontitis Generalized aggressive periodontitis Periodontitis as a manifestation of systemic disease Periodontosis Necrotizing periodontal diseases Abscesses of the periodontium Combined periodontic-endodontic lesions.

Oral Lichen Planus (OLP) | Desquamative Gingivitis | Continuing Education Course |

Int J Adv Case Rep ;2: The clinical gijgivitis of desquamative gingivitis. Squamous cell papilloma Keratoacanthoma Malignant: Periapical, mandibular and maxillary hard tissues — Bones of jaws Agnathia Alveolar osteitis Buccal exostosis Cherubism Idiopathic osteosclerosis Mandibular fracture Microgenia Micrognathia Intraosseous cysts Odontogenic: Nasopalatine duct Median mandibular Median palatal Traumatic bone Osteoma Osteomyelitis Osteonecrosis Bisphosphonate-associated Neuralgia-inducing cavitational desquamatjve Osteoradionecrosis Osteoporotic bone marrow defect Paget’s disease of bone Periapical abscess Phoenix abscess Periapical periodontitis Stafne defect Torus mandibularis.


There was widespread erythema and inflammation of maxillary anterior labial gingiva. Open in a separate window.

Erythematous gingiva with loss of stippling, extending apically from the gingival margins to the alveolar mucosa is a frequent observation. Retrieved from ” https: Arteaga and Eisenberg 36 Definitive diagnosis of DG should be made by incisional biopsy, histopathological examination and DIF. The definitive diagnosis can be made by histopathological, direct DIF and indirect immune fluorescent IIF examinations of the tissues obtained from the lesions, in addition to examination of autoantibodies in the circulation Nikolsky’s sign positive Click here to view.

The patient was asked to stop the topical steroid application and reinforcement of oral hygiene instructions were given. Limitation of oral function and speech difficulties due to pain can also be observed 26. Diagnostic pathways and clinical significance of desquamative gingivitis. There were no associated ocular, cutaneous or genital lesions.

Desquamative gingivitis: A review

Topical use of sicatrizing drugs as supportive treatment accelerates regression of lesions J Pharm Bioallied Sci. For generalized lesions, prednisolone mgbetamethasone 0.

Dilemma in diagnosis of mucocutaneous disorders. Br J Oral Maxillofac Surg.

N Engl J Med. However, the oral lesions usually heal without scarring. Published online Apr 1. Users may do so gingigitis any gingivtis manner, but not in any way that suggests the journal endorses its use. Intraoral restorations or prosthesis should be removed 8. Desquamative gingivitis involves lesions of the free and attached gingiva. Esophageal strictures may lead to dysphagia and may rarely rupture to result in mediastinitis.


By using this site, you agree to the Terms of Use and Privacy Policy. Chronic diffuse desquamative gingivitis.

Ultrastructural study of mechanobullous desquamative gingivitis: W B Saunders; Conventional therapy Debridement Scaling and root planing Full mouth disinfection Full mouth ultrasonic debridement.

Several mucocutaneous diseases in which clinical desquamative gingivitis is observed have been reported in the literature Table 1. Other topical treatments may include topical tacrolimus or topical pimecrolimus.

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The lesions showed considerable improvement after steroid application. Histopathological features in the present case, showed subepithelial cleft and basal cell degeneration, along with band of intense chronic inflammatory cell infiltrate consisting predominantly gingivitsi plasma cells and areas of hemorrhage. The condition belongs to a group of mucocutaneous autoimmune blistering disorders often collectively referred to as subepithelial bullous dermatoses.

In general, there is chronic pain, which especially increases with the intake of acidic foods.